install
+
Camera: iPhone 4S
Aperture: f/2.4
Exposure: 1/20th
Focal Length: 4mm
Exif sexpigeon:

Encourage your players to wear leather and mirrorshades.

sexpigeon:

Encourage your players to wear leather and mirrorshades.

Posted 1 day ago.
+ kicker-of-elves:


Dinka men in Sudan     National Geographic November 1984   Angela Fisher




they’re wearing hand-beaded corsets, just fyi.  the dinka have incredible clothing

kicker-of-elves:

Dinka men in Sudan     National Geographic November 1984   Angela Fisher

they’re wearing hand-beaded corsets, just fyi. the dinka have incredible clothing

Posted 1 day ago.
+
Posted 1 day ago. Tagged with Show all posts tagged with "admired".admired, .
here is some advice about menstrual cramps: take lots of aspirin
i found this out by trial and error, then wikipedia to confirm the theory behind it.  it turns out one of aspirin’s mechanisms of pain relief is to block the production of the hormone that causes muscle cramping, preventing cramps before they start.  wikipedia:

Aspirin’s ability to suppress the production of prostaglandins and thromboxanes is due to its irreversible inactivation of the cyclooxygenase(PTGS) enzyme required for prostaglandin and thromboxane synthesis. Aspirin acts as an acetylating agent where an acetyl group is covalently attached to a serine residue in the active site of the PTGS enzyme. This makes aspirin different from other NSAIDs (such as diclofenac and ibuprofen), which are reversible inhibitors.
Low-dose, long-term aspirin use irreversibly blocks the formation of thromboxane A2 in platelets, producing an inhibitory effect on platelet aggregation. This antithrombotic property makes aspirin useful for reducing the incidence of heart attacks.[110]40 mg of aspirin a day is able to inhibit a large proportion of maximum thromboxane A2 release provoked acutely, with the prostaglandin I2 synthesis being little affected; however, higher doses of aspirin are required to attain further inhibition.[111]
Prostaglandins, local hormones produced in the body, have diverse effects, including the transmission of pain information to the brain, modulation of the hypothalamic thermostat, and inflammation. Thromboxanes are responsible for the aggregation of platelets that form blood clots. Heart attacks are caused primarily by blood clots, and low doses of aspirin are seen as an effective medical intervention for acute myocardial infarction. An unwanted side effect of the effective anticlotting action of aspirin is that it may cause excessive bleeding.

EMPHASIS MINE.  
what is this hormone? prostaglandin.  it’s the shit your body makes to cause your uterus to spasm, which expels the blood and tissue that has built up on the walls during the menstrual cycle. wikipedia again:

They are mediators and have a variety of strong physiological effects, such as regulating the contraction and relaxation of smooth muscle tissue.[1] Prostaglandins are not endocrine hormones, but autocrine or paracrine, which are locally acting messenger molecules. They differ from hormones in that they are not produced at a discrete site but in many places throughout the human body. Also, their target cells are present in the immediate vicinity of the site of theirsecretion(of which there are many).
The prostaglandins, together with the thromboxanes and prostacyclins, form the prostanoid class of fatty acid derivatives, a subclass of eicosanoids.

sounds serious.  what’s the mechanism involved?  take us away, mayo clinic:

During menstrual periods, your uterus contracts to help expel its lining. Hormone-like substances (prostaglandins) involved in pain and inflammation trigger the uterine muscle contractions. Higher levels of prostaglandins are associated with more severe menstrual cramps.
Many experts believe that severe contractions constrict the blood vessels feeding the uterus. The resulting pain can be compared to the angina that occurs when blocked coronary arteries starve portions of the heart of food and oxygen.

the part wikipedia mentioned earlier about aspirin being a blood thinner as well as a prostaglandin blocker is important here because aspirin is lauching a two-part attack on the mechanisms that cause menstrual cramping specifically: 
uterine contractions caused by prostaglandin
lack of blood flow to the uterus itself caused by vessel constriction
i have sicknasty cramps.  they do not respond to anything, not even weed.  we’re talking rolling-involuntarily-on-the-ground-in-a-fetal-position-level cramps.  8 on the pain scale, where a 10 is uncontrollable screaming.  aspirin, between 4-6 of them at the first sign of cramping and doses of 1-2 as bumpers until the cramping part of the period ends, is the only effective treatment i have ever found.  
so if you have a uterus and cramping problems, and you do not have any sort of ulcers, aspirin sensitivity, bleeding or clotting problems, or are on medications that could potentially be disrupted by aspirin, please give it a shot.  i just take plain, uncoated, generic-ass aspirin.  if you don’t have a uterus or dont menstruate but know people who do, pass this on.  please include warnings about potential complications for anyone with aspirin sensitivities due to health, genetics or medication.
edit: for some fucking reason, Midol and other menstrual-specific preparations do not appear to actually contain any fucking aspirin.  Midol for example is primarily paracetamol, which does not as far as i can tell affect prostaglandin production in the least and is just a mild analgesic/anti-inflammatory.  so, fuck everything marketed to women, i guess is the lesson here.

here is some advice about menstrual cramps: take lots of aspirin


i found this out by trial and error, then wikipedia to confirm the theory behind it.  it turns out one of aspirin’s mechanisms of pain relief is to block the production of the hormone that causes muscle cramping, preventing cramps before they start.  wikipedia:

Aspirin’s ability to suppress the production of prostaglandins and thromboxanes is due to its irreversible inactivation of the cyclooxygenase(PTGS) enzyme required for prostaglandin and thromboxane synthesis. Aspirin acts as an acetylating agent where an acetyl group is covalently attached to a serine residue in the active site of the PTGS enzyme. This makes aspirin different from other NSAIDs (such as diclofenac and ibuprofen), which are reversible inhibitors.

Low-dose, long-term aspirin use irreversibly blocks the formation of thromboxane A2 in platelets, producing an inhibitory effect on platelet aggregation. This antithrombotic property makes aspirin useful for reducing the incidence of heart attacks.[110]40 mg of aspirin a day is able to inhibit a large proportion of maximum thromboxane Arelease provoked acutely, with the prostaglandin I2 synthesis being little affected; however, higher doses of aspirin are required to attain further inhibition.[111]

Prostaglandins, local hormones produced in the body, have diverse effects, including the transmission of pain information to the brain, modulation of the hypothalamic thermostat, and inflammation. Thromboxanes are responsible for the aggregation of platelets that form blood clots. Heart attacks are caused primarily by blood clots, and low doses of aspirin are seen as an effective medical intervention for acute myocardial infarction. An unwanted side effect of the effective anticlotting action of aspirin is that it may cause excessive bleeding.

EMPHASIS MINE.  

what is this hormone? prostaglandin.  it’s the shit your body makes to cause your uterus to spasm, which expels the blood and tissue that has built up on the walls during the menstrual cycle. wikipedia again:

They are mediators and have a variety of strong physiological effects, such as regulating the contraction and relaxation of smooth muscle tissue.[1] Prostaglandins are not endocrine hormones, but autocrine or paracrine, which are locally acting messenger molecules. They differ from hormones in that they are not produced at a discrete site but in many places throughout the human body. Also, their target cells are present in the immediate vicinity of the site of theirsecretion(of which there are many).

The prostaglandins, together with the thromboxanes and prostacyclins, form the prostanoid class of fatty acid derivatives, a subclass of eicosanoids.

sounds serious.  what’s the mechanism involved?  take us away, mayo clinic:

During menstrual periods, your uterus contracts to help expel its lining. Hormone-like substances (prostaglandins) involved in pain and inflammation trigger the uterine muscle contractions. Higher levels of prostaglandins are associated with more severe menstrual cramps.

Many experts believe that severe contractions constrict the blood vessels feeding the uterus. The resulting pain can be compared to the angina that occurs when blocked coronary arteries starve portions of the heart of food and oxygen.

the part wikipedia mentioned earlier about aspirin being a blood thinner as well as a prostaglandin blocker is important here because aspirin is lauching a two-part attack on the mechanisms that cause menstrual cramping specifically: 

  1. uterine contractions caused by prostaglandin
  2. lack of blood flow to the uterus itself caused by vessel constriction

i have sicknasty cramps.  they do not respond to anything, not even weed.  we’re talking rolling-involuntarily-on-the-ground-in-a-fetal-position-level cramps.  8 on the pain scale, where a 10 is uncontrollable screaming.  aspirin, between 4-6 of them at the first sign of cramping and doses of 1-2 as bumpers until the cramping part of the period ends, is the only effective treatment i have ever found.  

so if you have a uterus and cramping problems, and you do not have any sort of ulcers, aspirin sensitivity, bleeding or clotting problems, or are on medications that could potentially be disrupted by aspirin, please give it a shot.  i just take plain, uncoated, generic-ass aspirin.  if you don’t have a uterus or dont menstruate but know people who do, pass this on.  please include warnings about potential complications for anyone with aspirin sensitivities due to health, genetics or medication.

edit: for some fucking reason, Midol and other menstrual-specific preparations do not appear to actually contain any fucking aspirin.  Midol for example is primarily paracetamol, which does not as far as i can tell affect prostaglandin production in the least and is just a mild analgesic/anti-inflammatory.  so, fuck everything marketed to women, i guess is the lesson here.

i still like you!

just a heads up: i am cleaning up my dash, which has become a Tumblr Saviour wasteland.  

this means i am aggressively unfollowing people, even people i love, who post untagged fandom stuff i dont care about, large numbers of posts with >10k notes (i’ve generally seen them! a few times! thank you!), extensive tumbleft argue/callouts, lots of untagged tmi tuesdays, lots of untagged ask memes, click here to sign petition to convince sociopathic plutocrat to do a thing out of the goodness of his heart, please donate to such and such a thing or cause which i cannot do because i’m fucking broke, and/or constant and unrelenting self-criticism*.

all this shit is exhausting to me when i’m just trying to look at reference photos, paintings, read essays, etc.  and yall should be allowed to post whatever the heck you want on your own god damned blog without me squinting and scrolling so resentfully hard that i get a headache + wheelburn.

this applies to facebook, tumblr and twitter.

thank you for understanding, i’m not mad at you, and please keep in touch via PM or IM or whatever else; i swear to god i still like you as a person

(* think about it: if you saw someone on your dash constantly talking shit about your friends you would probably want to scream at them and/or unfollow, right? right )

Posted 2 days ago. Tagged with Show all posts tagged with "blog".blog, .

3liza:

krystal + junk
digital
2013
for hail gay satan zine

characters from “hail gay satan” by partydog/lamezone

buy zine here

wipgif:

diurnal reblog because i added a wipgif. i read my contributor copy today and it is fucking incredible. and there are some other guest art pieces in HGS that are STUNNING; they put this one to shame.

Posted 2 days ago.

write a chrome extension that seamlessly slots rss feeds into your tumblr dash

+ ahiddengardenofsecrets:

A new time has raised…
Gertrude Hubbell, Ruth Peters and Mildred Grimwood, hiking their skirts at the shoreline of the beach in Averne, Queens, New York, NY
photo by Wallace G. Levison, September 8, 1897

ahiddengardenofsecrets:

A new time has raised…

Gertrude Hubbell, Ruth Peters and Mildred Grimwood, hiking their skirts at the shoreline of the beach in Averne, Queens, New York, NY

photo by Wallace G. Levison, September 8, 1897

Posted 2 days ago.
+ saintetiennedemermorte:

Franz von Stuck.

saintetiennedemermorte:

Franz von Stuck.

Posted 2 days ago. Tagged with Show all posts tagged with "admired".admired, .
theincompletenesstheorem:

Franz von Stuck, The Kiss of the Sphinx, c. 1895

theincompletenesstheorem:

Franz von Stuck, 
The Kiss of the Sphinx, c. 1895

Posted 2 days ago. Tagged with Show all posts tagged with "admired".admired, .
+ Kiss of the Sphynx1895Franz von Stuck

Kiss of the Sphynx
1895
Franz von Stuck

+ faustyflakes:

Captain George K.H. Coussmaker, Captain and Lieutenant of the 1st Foot Guards.  Sir Joshua Reynolds, 1782. Detail.

faustyflakes:

Captain George K.H. Coussmaker, Captain and Lieutenant of the 1st Foot Guards.  Sir Joshua Reynolds, 1782. Detail.

Posted 2 days ago. Tagged with Show all posts tagged with "admired".admired, .
+ jerkcity:

#5241: one is too small

jerkcity:

#5241: one is too small

Posted 3 days ago.

krystal + junk
digital
2013
for hail gay satan zine

characters from “hail gay satan” by partydog/lamezone

buy zine here

wipgif:

lamezone:

ON SALE NOW!

LAMEZONE COLLECTION TWO

four things!

HAIL GAY SATAN- non-narrative gay demon atmosphere (45pgs)
THE BODY IS A SYSTEM- haunted vhs horror (26pgs)
SELECTED ffff.- early work (22pgs)
ASSORTED GARBAGES no. 2 - sketchbook pages (44pgs)
plus guest gay satan art from 11 rad contributors!!!! wow

i’m in this!